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Cystin, a novel cilia-associated protein, is disrupted in the cpk mouse model of polycystic kidney disease
The congenital polycystic kidney (cpk) mutation is the most extensively characterized mouse model of polycystic kidney disease (PKD). The renal cystic disease is fully expressed in homozygotes and is strikingly similar to human autosomal recessive PKD (ARPKD), whereas genetic background modulates th...
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| 主要な著者: | , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Clinical Investigation
2002
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC150876/ https://ncbi.nlm.nih.gov/pubmed/11854326 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI14099 |
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