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TIMP-2 upregulates RECK expression via dephosphorylation of paxillin tyrosine residues 31 and 118
We previously demonstrated that TIMP-2 increases the association of Crk with C3G and via subsequent activation of Rap1 enhances the expression of RECK, a membrane-anchored MMP inhibitor. In the present study, we investigate the mechanism of how the TIMP-2 signal is transduced from the α3β1 integrin...
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| Главные авторы: | , , , , , |
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| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
2006
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1502148/ https://ncbi.nlm.nih.gov/pubmed/16491114 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.onc.1209444 |
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