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Abasic site binding by the human apurinic endonuclease, Ape, and determination of the DNA contact sites.

The mutagenic and lethal effects of abasic sites in DNA are averted by repair initiated by 'class II' apurinic (AP) endonucleases, which cleave immediately 5'to abasic sites. We examined substrate binding by the human AP endonuclease, Ape protein (also called Hap1, Apex or Ref-1). In electrophoretic...

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Uloženo v:
Vydáno v:Nucleic Acids Res
Hlavní autoři: Wilson, D M, Takeshita, M, Demple, B
Médium: Artigo
Jazyk:Inglês
Vydáno: Oxford University Press 1997
Témata:
Research Article
On-line přístup:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC146540/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9023101/
https://ncbi.nlm.nih.govhttps://doi.org/10.1093/nar/25.5.933
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