Dominant mutations in three different subunits of replication factor C suppress replication defects in yeast PCNA mutants.

To identify proteins that interact with the yeast proliferating cell nuclear antigen (PCNA), we used a genetic approach to isolate mutations that compensate for the defects in cold-sensitive (Cs(-)) mutants of yeast PCNA (POL30). Because the cocrystal structure of human PCNA and a p21(WAF1/CIP1) pep...

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Main Authors: Amin, N S, Tuffo, K M, Holm, C
Formato: Artigo
Idioma:Inglês
Publicado: 1999
Assuntos:
Research Article
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC1460869/
https://ncbi.nlm.nih.gov/pubmed/10581271
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