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Thalidomide attenuates nitric oxide mediated angiogenesis by blocking migration of endothelial cells
BACKGROUND: Thalidomide is an immunomodulatory agent, which arrests angiogenesis. The mechanism of anti-angiogenic activity of thalidomide is not fully understood. As nitric oxide is involved in angiogenesis, we speculate a cross-talk between thalidomide and nitric oxide signaling pathway to define...
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| 主要な著者: | , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2006
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1456963/ https://ncbi.nlm.nih.gov/pubmed/16584574 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2121-7-17 |
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