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Senescence-associated phenotypes in Akita diabetic mice are enhanced by absence of bradykinin B2 receptors

We have previously reported that genetically increased angiotensin-converting enzyme levels, or absence of the bradykinin B2 receptor, increase kidney damage in diabetic mice. We demonstrate here that this is part of a more general phenomenon — diabetes and, to a lesser degree, absence of the B2 rec...

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Detalhes bibliográficos
Main Authors: Kakoki, Masao, Kizer, Catherine M., Yi, Xianwen, Takahashi, Nobuyuki, Kim, Hyung-Suk, Bagnell, C. Robert, Edgell, Cora-Jean S., Maeda, Nobuyo, Jennette, J. Charles, Smithies, Oliver
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 2006
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1430357/
https://ncbi.nlm.nih.gov/pubmed/16604193
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI26958
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