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Activation of Rac1 by shear stress in endothelial cells mediates both cytoskeletal reorganization and effects on gene expression

Hemodynamic shear stress is a fundamental determinant of vascular remodeling and atherogenesis. Changes in focal adhesions, cytoskeletal organization and gene expression are major responses of endothelial cells to shear stress. Here, we show that activation of the small GTPase Rac is essential for g...

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Bibliografski detalji
Glavni autori: Tzima, Eleni, Del Pozo, Miguel Angel, Kiosses, William B., Mohamed, Samih A., Li, Song, Chien, Shu, Schwartz, Martin Alexander
Format: Artigo
Jezik:Inglês
Izdano: Oxford University Press 2002
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC139108/
https://ncbi.nlm.nih.gov/pubmed/12486000
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/cdf688
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