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Activation of Rac1 by shear stress in endothelial cells mediates both cytoskeletal reorganization and effects on gene expression

Hemodynamic shear stress is a fundamental determinant of vascular remodeling and atherogenesis. Changes in focal adhesions, cytoskeletal organization and gene expression are major responses of endothelial cells to shear stress. Here, we show that activation of the small GTPase Rac is essential for g...

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Detalhes bibliográficos
Main Authors: Tzima, Eleni, Del Pozo, Miguel Angel, Kiosses, William B., Mohamed, Samih A., Li, Song, Chien, Shu, Schwartz, Martin Alexander
Formato: Artigo
Idioma:Inglês
Publicado em: Oxford University Press 2002
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC139108/
https://ncbi.nlm.nih.gov/pubmed/12486000
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/cdf688
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