Ventricular septal defect and cardiomyopathy in mice lacking the transcription factor CHF1/Hey2
Ventricular septal defects are common in human infants, but the genetic programs that control ventricular septation are poorly understood. Here we report that mice with a targeted disruption of the cardiovascular basic helix–loop–helix factor (CHF)1/Hey2 gene show isolated ventricular septal defects...
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| Veröffentlicht in: | Proc Natl Acad Sci U S A |
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| Hauptverfasser: | , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
National Academy of Sciences
2002
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| Schlagworte: | |
| Online-Zugang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC138588/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12454287/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.252648999 |
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