Ventricular septal defect and cardiomyopathy in mice lacking the transcription factor CHF1/Hey2
Ventricular septal defects are common in human infants, but the genetic programs that control ventricular septation are poorly understood. Here we report that mice with a targeted disruption of the cardiovascular basic helix–loop–helix factor (CHF)1/Hey2 gene show isolated ventricular septal defects...
Furkejuvvon:
| Publikašuvnnas: | Proc Natl Acad Sci U S A |
|---|---|
| Váldodahkkit: | , , , , , |
| Materiálatiipa: | Artigo |
| Giella: | Inglês |
| Almmustuhtton: |
National Academy of Sciences
2002
|
| Fáttát: | |
| Liŋkkat: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC138588/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12454287/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.252648999 |
| Fáddágilkorat: |
Eai fáddágilkorat, Lasit vuosttaš fáddágilkora!
|