Impaired inflammatory pain and thermal hyperalgesia in mice expressing neuron-specific dominant negative mitogen activated protein kinase kinase (MEK)

Lignende værker

• Metabotropic Glutamate Receptor Subtypes 1 and 5 Are Activators of Extracellular Signal-Regulated Kinase Signaling Required for Inflammatory Pain in Mice
  af: Karim, Farzana, et al.
  Udgivet: (2001)
• Nuclear translocation of mitogen-activated protein kinase kinase (MEK1) in response to mitogenic stimulation
  af: Jaaro, Hanna, et al.
  Udgivet: (1997)
• MEK-1 phosphorylation by MEK kinase, Raf, and mitogen-activated protein kinase: analysis of phosphopeptides and regulation of activity.
  af: Gardner, A M, et al.
  Udgivet: (1994)
• Prostaglandin and Protein Kinase A-Dependent Modulation of Vanilloid Receptor Function by Metabotropic Glutamate Receptor 5: Potential Mechanism for Thermal Hyperalgesia
  af: Hu, Hui-Juan, et al.
  Udgivet: (2002)
• Reduced GABAergic transmission in the ventrobasal thalamus contributes to thermal hyperalgesia in chronic inflammatory pain
  af: Zhang, Chan, et al.
  Udgivet: (2017)