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Bcr–Abl activates the AKT/FoxO3 signalling pathway to restrict transforming growth factor-β-mediated cytostatic signals

The fusion of Abl with either Bcr or Tel in human leukaemia leads to the constitutive activation of Abl tyrosine kinase, which in turn induces growth-factor-independent proliferation and cell survival. However, the mechanism by which Bcr–Abl induces cellular transformation has not yet been well char...

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Detalhes bibliográficos
Main Authors: Atfi, Azeddine, Abécassis, Lucile, Bourgeade, Marie-Francoise
Formato: Artigo
Idioma:Inglês
Publicado em: 2005
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1369182/
https://ncbi.nlm.nih.gov/pubmed/16113647
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.embor.7400501
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