Deacetylation of the Herpes Simplex Virus Type 1 Latency-Associated Transcript (LAT) Enhancer and a Decrease in LAT Abundance Precede an Increase in ICP0 Transcriptional Permissiveness at Early Times Postexplant

Antzeko izenburuak

• The Herpes Simplex Virus Type 1 Latency-Associated Transcript (LAT) Enhancer/rcr Is Hyperacetylated during Latency Independently of LAT Transcription
  nork: Kubat, Nicole J., et al.
  Argitaratua: (2004)
• Tissue-Specific Splicing of the Herpes Simplex Virus Type 1 Latency-Associated Transcript (LAT) Intron in LAT Transgenic Mice
  nork: Gussow, Anne M., et al.
  Argitaratua: (2006)
• Novel Less-Abundant Viral MicroRNAs Encoded by Herpes Simplex Virus 2 Latency-Associated Transcript and Their Roles in Regulating ICP34.5 and ICP0 mRNAs
  nork: Tang, Shuang, et al.
  Argitaratua: (2009)
• During Herpes Simplex Virus Type 1 Infection of Rabbits, the Ability To Express the Latency-Associated Transcript Increases Latent-Phase Transcription of Lytic Genes
  nork: Giordani, Nicole V., et al.
  Argitaratua: (2008)
• Transcriptional Regulation of Latency-Associated Transcripts (LATs) of Herpes Simplex Viruses
  nork: Zhang, Ying, et al.
  Argitaratua: (2020)