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Loss of retinoblastoma but not p16 function allows bypass of replicative senescence in human fibroblasts
Current models envision replicative senescence to be under dual control by the p53 and retinoblastoma (RB) tumour-suppressor pathways. The role of the p16(INK4a)–RB pathway is controversial, and the function of RB in human cells has not been tested directly. We used targeted homologous recombination...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2003
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1326371/ https://ncbi.nlm.nih.gov/pubmed/14566323 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.embor.7400001 |
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