Tumour necrosis factor-induced activation of c-Jun N-terminal kinase is sensitive to caspase-dependent modulation while activation of mitogen-activated protein kinase (MAPK) or p38 MAPK is not.

Lignende værker

• Type II tumour necrosis factor-alpha receptor (TNFR2) activates c-Jun N-terminal kinase (JNK) but not mitogen-activated protein kinase (MAPK) or p38 MAPK pathways.
  af: Jupp, O J, et al.
  Udgivet: (2001)
• Distinct regulation of cytosolic phospholipase A2 phosphorylation, translocation, proteolysis and activation by tumour necrosis factor-receptor subtypes.
  af: Jupp, Orla J, et al.
  Udgivet: (2003)
• Role of caspases in dexamethasone-induced apoptosis and activation of c-Jun NH(2)-terminal kinase and p38 mitogen-activated protein kinase in human eosinophils
  af: Zhang, J P, et al.
  Udgivet: (2000)
• Opposing Effects of Jun Kinase and p38 Mitogen-Activated Protein Kinases on Cardiomyocyte Hypertrophy
  af: Nemoto, Shino, et al.
  Udgivet: (1998)
• The Mitogen-Activated Protein Kinase (MAPK)-Activated Protein Kinases MK2 and MK3 Cooperate in Stimulation of Tumor Necrosis Factor Biosynthesis and Stabilization of p38 MAPK
  af: Ronkina, N., et al.
  Udgivet: (2007)