Loss of Pin1 function in the mouse causes phenotypes resembling cyclin D1-null phenotypes

Phosphorylation of proteins on serine/threonine residues preceding proline is a key signaling mechanism. The conformation and function of a subset of these phosphorylated proteins is regulated by the prolyl isomerase Pin1 through isomerization of phosphorylated Ser/Thr-Pro bonds. Although young Pin1...

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Detaylı Bibliyografya
Asıl Yazarlar: Liou, Yih-Cherng, Ryo, Akihide, Huang, Han-Kuei, Lu, Pei-Jung, Bronson, Roderick, Fujimori, Fumihiro, Uchida, Takafumi, Hunter, Tony, Lu, Kun Ping
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: The National Academy of Sciences 2002
Konular:
Biological Sciences
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC122191/
https://ncbi.nlm.nih.gov/pubmed/11805292
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.032404099
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