Bcl-xL overexpression attenuates glutathione depletion in FL5.12 cells following interleukin-3 withdrawal.

Bcl-xL and bax are bcl-2-related genes whose protein products either inhibit or promote apoptosis. Oxidative damage, including the loss of glutathione, has been implicated in the induction of apoptosis. The ability of the Bcl proteins to affect GSH was assessed in control, bax- and bcl-xL-transfecte...

Full description

Saved in:
Bibliographic Details
Main Authors: Bojes, H K, Datta, K, Xu, J, Chin, A, Simonian, P, Nuñez, G, Kehrer, J P
Format: Artigo
Language:Inglês
Published: 1997
Subjects:
Research Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC1218562/
https://ncbi.nlm.nih.gov/pubmed/9230108
Tags: Add Tag
No Tags, Be the first to tag this record!

Similar Items