Bcl-xL overexpression attenuates glutathione depletion in FL5.12 cells following interleukin-3 withdrawal.

Bcl-xL and bax are bcl-2-related genes whose protein products either inhibit or promote apoptosis. Oxidative damage, including the loss of glutathione, has been implicated in the induction of apoptosis. The ability of the Bcl proteins to affect GSH was assessed in control, bax- and bcl-xL-transfecte...

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Bibliographische Detailangaben
Hauptverfasser: Bojes, H K, Datta, K, Xu, J, Chin, A, Simonian, P, Nuñez, G, Kehrer, J P
Format: Artigo
Sprache:Inglês
Veröffentlicht: 1997
Schlagworte:
Research Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1218562/
https://ncbi.nlm.nih.gov/pubmed/9230108
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