ATP depletion inhibits glucocorticoid-induced thymocyte apoptosis.

In quiescent thymocytes, mitochondrial de-energization was not correlated to apoptotic death. In fact, thymocytes treated with oligomycin, a highly specific inhibitor of ATP synthase, alone or with atractyloside to block ATP translocation from the cytoplasm, were alive, even if their mitochondria we...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Stefanelli, C, Bonavita, F, Stanic', I, Farruggia, G, Falcieri, E, Robuffo, I, Pignatti, C, Muscari, C, Rossoni, C, Guarnieri, C, Caldarera, C M
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: 1997
Θέματα:
Research Article
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC1218274/
https://ncbi.nlm.nih.gov/pubmed/9148768
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