Altered axonal architecture by removal of the heavily phosphorylated neurofilament tail domains strongly slows superoxide dismutase 1 mutant-mediated ALS

פריטים דומים

• Gene replacement in mice reveals that the heavily phosphorylated tail of neurofilament heavy subunit does not affect axonal caliber or the transit of cargoes in slow axonal transport
  מאת: Rao, Mala V., et al.
  יצא לאור: (2002)
• Absence of neurofilaments reduces the selective vulnerability of motor neurons and slows disease caused by a familial amyotrophic lateral sclerosis-linked superoxide dismutase 1 mutant
  מאת: Williamson, Toni L., et al.
  יצא לאור: (1998)
• Protective effect of neurofilament heavy gene overexpression in motor neuron disease induced by mutant superoxide dismutase
  מאת: Couillard-Després, Sébastien, et al.
  יצא לאור: (1998)
• Neurofilament-dependent Radial Growth of Motor Axons and Axonal Organization of Neurofilaments Does Not Require the Neurofilament Heavy Subunit (NF-H) or Its Phosphorylation
  מאת: Rao, Mala V., et al.
  יצא לאור: (1998)
• Neurofilaments Are Transported Rapidly But Intermittently in Axons: Implications for Slow Axonal Transport
  מאת: Roy, Subhojit, et al.
  יצא לאור: (2000)