The Herpes Simplex Virus U(S)11 Protein Effectively Compensates for the γ(1)34.5 Gene if Present before Activation of Protein Kinase R by Precluding Its Phosphorylation and That of the α Subunit of Eukaryotic Translation Initiation Factor 2

Samankaltaisia teoksia

• The Second-Site Mutation in the Herpes Simplex Virus Recombinants Lacking the γ(1)34.5 Genes Precludes Shutoff of Protein Synthesis by Blocking the Phosphorylation of eIF-2α
  Tekijä: Cassady, Kevin A., et al.
  Julkaistu: (1998)
• The γ(1)34.5 protein of herpes simplex virus 1 complexes with protein phosphatase 1α to dephosphorylate the α subunit of the eukaryotic translation initiation factor 2 and preclude the shutoff of protein synthesis by double-stranded RNA-activated protein kinase
  Tekijä: He, Bin, et al.
  Julkaistu: (1997)
• Regulation of herpes simplex virus γ(1)34.5 expression and oncolysis of diffuse liver metastases by Myb34.5
  Tekijä: Nakamura, Hideo, et al.
  Julkaistu: (2002)
• The gamma 1(34.5) gene of herpes simplex virus 1 precludes neuroblastoma cells from triggering total shutoff of protein synthesis characteristic of programed cell death in neuronal cells.
  Tekijä: Chou, J, et al.
  Julkaistu: (1992)
• Second-Site Mutation Outside of the U(S)10-12 Domain of Δγ(1)34.5 Herpes Simplex Virus 1 Recombinant Blocks the Shutoff of Protein Synthesis Induced by Activated Protein Kinase R and Partially Restores Neurovirulence
  Tekijä: Cassady, Kevin A., et al.
  Julkaistu: (2002)