Requirement of Cyclin E-Cdk2 Inhibition in p16(INK4a)-Mediated Growth Suppression
Loss-of-function mutations of p16(INK4a) have been identified in a large number of human tumors. An established biochemical function of p16 is its ability to specifically inhibit cyclin D-dependent kinases in vitro, and this inhibition is believed to be the cause of the p16-mediated G(1) cell cycle...
Guardat en:
| Publicat a: | Mol Cell Biol |
|---|---|
| Autors principals: | , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Taylor & Francis
1998
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC109114/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9710613/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.18.9.5284 |
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