A Second-Site Mutation That Restores Replication of a Tat-Defective Human Immunodeficiency Virus
We previously constructed a large set of mutants of the human immunodeficiency virus type 1 (HIV-1) regulatory protein Tat with conservative amino acid substitutions in the activation domain. These Tat variants were analyzed in the context of the infectious virus, and several mutants were found to b...
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| Gepubliceerd in: | J Virol |
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| Hoofdauteurs: | , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Society for Microbiology (ASM)
1999
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC104035/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10074125/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.73.4.2781-2789.1999 |
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