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Cyclosporin treatment does not impair the release of nitric oxide in human coronary arteries.

OBJECTIVE--It has been hypothesised that compromised endothelial function can contribute to the toxic manifestations associated with cyclosporin therapy. In vitro animal studies have implicated inhibition of release of the endothelium derived relaxing factor, nitric oxide; however, this has not been...

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Detalhes bibliográficos
Main Authors: O'Neil, G S, Chester, A H, Kushwaha, S, Rose, M, Tadjkarimi, S, Yacoub, M H
Formato: Artigo
Idioma:Inglês
Publicado em: 1991
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1024646/
https://ncbi.nlm.nih.gov/pubmed/1718353
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