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Regulation of Cellular Signaling by CUL5 is Dependent on Its Neddylation Status

Background/Aims: CUL5 acts as the scaffold protein of the E3 ligase complex in the ubiquitin-dependent protein degradation pathways. Overexpression of CUL5 inhibits cellular proliferation, whereas inhibition of CUL5 expression induces proliferation and can lead to the development of clinical disorde...

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Principais autores: Skylar Sundquist, Sieun Ruth Lee, Alyssa Johnson, Haley M. Lynch, Abbey Schnell, Sarah Wieskamp, Henry N. Peabody, Lauren Kennington, Caroline Askonas, Charlotte Turner, David Hayes, Michael Hledin, Lauren Lee, Maria A. Burnatowska-Hledin
Formato: Artigo
Idioma:Inglês
Publicado em: Cell Physiol Biochem Press GmbH & Co KG 2026-03-01
coleção:Cellular Physiology and Biochemistry
Assuntos:
cul5
cellular growth
protein modification
neddylation
Acesso em linha:https://cellphysiolbiochem.com/Articles/000855/index.html
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