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Regulation of Cellular Signaling by CUL5 is Dependent on Its Neddylation Status

Background/Aims: CUL5 acts as the scaffold protein of the E3 ligase complex in the ubiquitin-dependent protein degradation pathways. Overexpression of CUL5 inhibits cellular proliferation, whereas inhibition of CUL5 expression induces proliferation and can lead to the development of clinical disorde...

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Detaylı Bibliyografya
Asıl Yazarlar: Skylar Sundquist, Sieun Ruth Lee, Alyssa Johnson, Haley M. Lynch, Abbey Schnell, Sarah Wieskamp, Henry N. Peabody, Lauren Kennington, Caroline Askonas, Charlotte Turner, David Hayes, Michael Hledin, Lauren Lee, Maria A. Burnatowska-Hledin
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Cell Physiol Biochem Press GmbH & Co KG 2026-03-01
Seri Bilgileri:Cellular Physiology and Biochemistry
Konular:
Online Erişim:https://cellphysiolbiochem.com/Articles/000855/index.html
Etiketler: Etiketle
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