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Human macrophages differentially produce specific resolvin or leukotriene signals that depend on bacterial pathogenicity

Abstract Proinflammatory eicosanoids (prostaglandins and leukotrienes) and specialized pro-resolving mediators (SPM) are temporally regulated during infections. Here we show that human macrophage phenotypes biosynthesize unique lipid mediator signatures when exposed to pathogenic bacteria. E. coli a...

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Autori principali: Oliver Werz, Jana Gerstmeier, Stephania Libreros, Xavier De la Rosa, Markus Werner, Paul C. Norris, Nan Chiang, Charles N. Serhan
Natura: Artigo
Lingua:Inglês
Pubblicazione: Nature Portfolio 2018-01-01
Serie:Nature Communications
Accesso online:https://doi.org/10.1038/s41467-017-02538-5
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