Fecal bile acid dysmetabolism and reduced ursodeoxycholic acid correlate with novel microbial signatures in feline chronic kidney disease

BackgroundMicrobial-derived secondary bile acids (SBAs) are reabsorbed and sensed via host receptors modulating cellular inflammation and fibrosis. Feline chronic kidney disease (CKD) occurs with progressive renal inflammation and fibrosis, mirroring the disease pathophysiology of human CKD patients...

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Detaylı Bibliyografya
Asıl Yazarlar:	John C. Rowe, Stacie C. Summers, Jessica M. Quimby, Jenessa A. Winston
Materyal Türü:	Artigo
Dil:	Inglês
Baskı/Yayın Bilgisi:	Frontiers Media S.A. 2024-10-01
Seri Bilgileri:	Frontiers in Microbiology
Konular:	bile acid inducible (bai) operon chronic kidney disease (CKD) 7α-dehydroxylation Peptacetobacter hiranonis Oscillospirales ursodeoxycholic acid
Online Erişim:	https://www.frontiersin.org/articles/10.3389/fmicb.2024.1458090/full
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Fecal bile acid dysmetabolism and reduced ursodeoxycholic acid correlate with novel microbial signatures in feline chronic kidney disease

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