XPF-ERCC1 protects liver, kidney and blood homeostasis outside the canonical excision repair pathways.
Loss of the XPF-ERCC1 endonuclease causes a dramatic phenotype that results in progeroid features associated with liver, kidney and bone marrow dysfunction. As this nuclease is involved in multiple DNA repair transactions, it is plausible that this severe phenotype results from the simultaneous inac...
保存先:
| 主要な著者: | , |
|---|---|
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Public Library of Science (PLoS)
2020-04-01
|
| シリーズ: | PLoS Genetics |
| オンライン･アクセス: | https://doi.org/10.1371/journal.pgen.1008555 |
| タグ: |
タグなし, このレコードへの初めてのタグを付けませんか!
|