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Inhibition of Galectin-3 by ADSC-derived exosomes after cavernous nerve injury ameliorates erectile dysfunction by modulating oxidative stress and inflammatory profibrotic cascades

Abstract Background Cavernous nerve injury (CNI) is a prominent etiological factor in the development of erectile dysfunction (ED). Nevertheless, the underlying pathophysiological mechanisms of CNI-induced ED (CNI-ED) are not fully elucidated. Galectin-3 (Gal-3), an indicator of inflammation and fib...

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Principais autores: Ruiyu Li, Guanbo Wang, Chen Feng, Tongxiang Diao, Shuai Liu, Qiang Fu
Format: Artigo
Sprog:Inglês
Udgivet: BMC 2025-11-01
Serier:Stem Cell Research & Therapy
Fag:
Erectile dysfunction
Galectin-3
Adipose mesenchymal stem cell-derived exosomes
Inflammatory profibrotic cascades
Oxidative stress
Online adgang:https://doi.org/10.1186/s13287-025-04749-w
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