The TUDOR domain of SMN is an H3K79 me1 histone mark reader
The survival of motor neuron (SMN) protein is depleted in spinal muscular atrophy (SMA) pathology and herein defined as the first reader of histone H3 mono-methylated on lysine 79 through its central TUDOR domain. Spinal muscular atrophy is the leading genetic cause of infant mortality and results f...
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| Auteurs principaux: | , , , , , , , |
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| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
Life Science Alliance LLC
2023-06-01
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| Collection: | Life Science Alliance |
| Accès en ligne: | https://www.life-science-alliance.org/lookup/doi/10.26508/lsa.202201752 |
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