Past, Present, and Future of the Pathophysiological Model of the Basal Ganglia

Por Obeso, José A., Lanciego, José L.
Publicado em 2011

Functional Neuroanatomy of the Basal Ganglia

Por Lanciego, José L., Luquin, Natasha, Obeso, José A.
Publicado em 2012

Habitual behavior and dopamine cell vulnerability in Parkinson disease

Por Hernández, Ledia F., Redgrave, Peter, Obeso, José A.
Publicado no Front Neuroanat (2015)

Editorial: Parkinson's disease: cell vulnerability and disease progression

Por Blesa, Javier, Lanciego, Jose L., Obeso, Jose A.
Publicado no Front Neuroanat (2015)

Corrigendum: Habitual Behavior and Dopamine Cell Vulnerability in Parkinson Disease

Por Hernández, Ledia F., Redgrave, Peter, Obeso, José A.
Publicado no Front Neuroanat (2015)

In Vivo Growing of New Cell Colonies in a Portion of Bone Marrow: Potential Use for Indirect Cell Therapy

Por Manzanedo, Ana, Rodriguez, Fidel, Obeso, Jose A., Rodriguez, Manuel
Publicado no Cell Med (2010)

Selective neuronal vulnerability in Parkinson disease

Por Surmeier, D. James, Obeso, José A., Halliday, Glenda M.
Publicado no Nat Rev Neurosci (2017)

Parkinson's Disease Is Not Simply a Prion Disorder

Por Surmeier, D. James, Obeso, José A., Halliday, Glenda M.
Publicado no J Neurosci (2017)

Milestones in Research on the Pathophysiology of Parkinson’s Disease

Por Wichmann, Thomas, DeLong, Mahlon R., Guridi, Jorge, Obeso, Jose A.
Publicado no Mov Disord (2011)

Early Paradoxical Increase of Dopamine: A Neurochemical Study of Olfactory Bulb in Asymptomatic and Symptomatic MPTP Treated Monkeys

Por Pifl, Christian, Reither, Harald, del Rey, Natalia Lopez-Gonzalez, Cavada, Carmen, Obeso, Jose A., Blesa, Javier
Publicado no Front Neuroanat (2017)

Human striatal recordings reveal abnormal discharge of projection neurons in Parkinson’s disease

Por Singh, Arun, Mewes, Klaus, Gross, Robert E., DeLong, Mahlon R., Obeso, José A., Papa, Stella M.
Publicado no Proc Natl Acad Sci U S A (2016)

Inter-hemispheric asymmetry of nigrostriatal dopaminergic lesion: a possible compensatory mechanism in Parkinson’s disease

Por Blesa, Javier, Juri, Carlos, García-Cabezas, Miguel Á., Adánez, Rebeca, Sánchez-González, Miguel Á., Cavada, Carmen, Obeso, José A.
Publicado em 2011

Goal-directed and habitual control in the basal ganglia: implications for Parkinson’s disease

Por Redgrave, Peter, Rodriguez, Manuel, Smith, Yoland, Rodriguez-Oroz, Maria C., Lehericy, Stephane, Bergman, Hagai, Agid, Yves, DeLong, Mahlon R., Obeso, Jose A.
Publicado em 2010

Lesion of the Centromedian Thalamic Nucleus in MPTP-Treated Monkeys

Por Lanciego, Jose L., Rodríguez-Oroz, Maria C., Blesa, Francisco J., Alvarez-Erviti, Lydia, Guridi, Jorge, Barroso-Chinea, Pedro, Smith, Yoland, Obeso, Jose A.
Publicado no Mov Disord (2008)

Grey matter hypometabolism and atrophy in Parkinson’s disease with cognitive impairment: a two-step process

Por González-Redondo, Rafael, García-García, David, Clavero, Pedro, Gasca-Salas, Carmen, García-Eulate, Reyes, Zubieta, José L., Arbizu, Javier, Obeso, José A., Rodríguez-Oroz, María C.
Publicado no Brain (2014)

NMDA receptor GluN2A/GluN2B subunit ratio as synaptic trait of levodopa-induced dyskinesias: from experimental models to patients

Por Mellone, Manuela, Stanic, Jennifer, Hernandez, Ledia F., Iglesias, Elena, Zianni, Elisa, Longhi, Annalisa, Prigent, Annick, Picconi, Barbara, Calabresi, Paolo, Hirsch, Etienne C., Obeso, Jose A., Di Luca, Monica, Gardoni, Fabrizio
Publicado no Front Cell Neurosci (2015)