Reduced P53 levels ameliorate neuromuscular junction loss without affecting motor neuron pathology in a mouse model of spinal muscular atrophy

von Courtney, Natalie L., Mole, Alannah J., Thomson, Alison K., Murray, Lyndsay M.
Veröffentlicht in Cell Death Dis (2019)

Synaptic withdrawal following nerve injury is influenced by postnatal maturity, muscle‐specific properties, and the presence of underlying pathology in mice

von Mole, Alannah J., Bell, Sarah, Thomson, Alison K., Dissanayake, Kosala N., Ribchester, Richard R., Murray, Lyndsay M.
Veröffentlicht in J Anat (2020)

Altered mitochondrial bioenergetics are responsible for the delay in Wallerian degeneration observed in neonatal mice

von Kline, Rachel A., Dissanayake, Kosala N., Hurtado, Maica Llavero, Martínez, Nicolás W., Ahl, Alexander, Mole, Alannah J., Lamont, Douglas J., Court, Felipe A., Ribchester, Richard R., Wishart, Thomas M., Murray, Lyndsay M.
Veröffentlicht in Neurobiol Dis (2019)

Abnormal fatty acid metabolism is a core component of spinal muscular atrophy

von Deguise, Marc‐Olivier, Baranello, Giovanni, Mastella, Chiara, Beauvais, Ariane, Michaud, Jean, Leone, Alessandro, De Amicis, Ramona, Battezzati, Alberto, Dunham, Christopher, Selby, Kathryn, Warman Chardon, Jodi, McMillan, Hugh J., Huang, Yu‐Ting, Courtney, Natalie L., Mole, Alannah J., Kubinski, Sabrina, Claus, Peter, Murray, Lyndsay M., Bowerman, Melissa, Gillingwater, Thomas H., Bertoli, Simona, Parson, Simon H., Kothary, Rashmi
Veröffentlicht in Ann Clin Transl Neurol (2019)