Lack of secondary structure characterizes the 5′ ends of mammalian mitochondrial mRNAs

Por Jones, Christie N., Wilkinson, Kevin A., Hung, Kimberly T., Weeks, Kevin M., Spremulli, Linda L.
Publicado em 2008

A Disease-causing Point Mutation in Human Mitochondrial tRNA(Met) Results in tRNA Misfolding Leading to Defects in Translational Initiation and Elongation

Por Jones, Christie N., Jones, Christopher I., Graham, William D., Agris, Paul F., Spremulli, Linda L.
Publicado em 2008

Analysis of the Functional Consequences of Lethal Mutations in Mitochondrial Translational Elongation Factors

Por Akama, Kenta, Christian, Brooke E., Jones, Christie N., Ueda, Takuya, Takeuchi, Nono, Spremulli, Linda L.
Publicado em 2010

Native Tertiary Structure and Nucleoside Modifications Suppress tRNA’s Intrinsic Ability to Activate the Innate Immune Sensor PKR

Por Nallagatla, Subba Rao, Jones, Christie N., Ghosh, Saikat Kumar B., Sharma, Suresh D., Cameron, Craig E., Spremulli, Linda L., Bevilacqua, Philip C.
Publicado em 2013

The human mitochondrial tRNA(Met): Structure/function relationship of a unique modification in the decoding of unconventional codons

Por Bilbille, Yann, Gustilo, Estella M., Harris, Kimberly A., Jones, Christie N., Lusic, Hrvoje, Kaiser, Robert J., Delaney, Michael O., Spremulli, Linda L., Deiters, Alexander, Agris, Paul F.
Publicado em 2010