SH1 domain autophosphorylation of P210 BCR/ABL is required for transformation but not growth factor independence.

Por Pendergast, A M, Gishizky, M L, Havlik, M H, Witte, O N
Publicado em 1993

A limited set of SH2 domains binds BCR through a high-affinity phosphotyrosine-independent interaction.

Por Muller, A J, Pendergast, A M, Havlik, M H, Puil, L, Pawson, T, Witte, O N
Publicado em 1992

En bloc substitution of the Src homology region 2 domain activates the transforming potential of the c-Abl protein tyrosine kinase.

Por Muller, A J, Pendergast, A M, Parmar, K, Havlik, M H, Rosenberg, N, Witte, O N
Publicado em 1993

Evidence for regulation of the human ABL tyrosine kinase by a cellular inhibitor.

Por Pendergast, A M, Muller, A J, Havlik, M H, Clark, R, McCormick, F, Witte, O N
Publicado em 1991