Endothelial dysfunction in the coronary microcirculation: a new clinical entity or an experimental finding?

Por Harrison, D G
Publicado em 1993

Cellular and molecular mechanisms of endothelial cell dysfunction.

Por Harrison, D G
Publicado em 1997

Insulin and the arginine paradox.

Por Kurz, S, Harrison, D G
Publicado em 1997

The calcium content of human erythrocytes

Por Harrison, D. G., Long, C.
Publicado em 1968

eNOS-overexpressing mice: too much NO makes the blood pressure low.

Por Drummond, G R, Harrison, D G
Publicado em 1998

Macrophages come to mind as keys to cognitive decline

Por Harrison, D.G., Guzik, Tomasz J.
Publicado no J Clin Invest (2016)

Hypercholesterolemia increases endothelial superoxide anion production.

Por Ohara, Y, Peterson, T E, Harrison, D G
Publicado em 1993

Evidence for a role of endothelin 1 and protein kinase C in nitroglycerin tolerance.

Por Münzel, T, Giaid, A, Kurz, S, Stewart, D J, Harrison, D G
Publicado em 1995

Reactive oxygen species produced by macrophage-derived foam cells regulate the activity of vascular matrix metalloproteinases in vitro. Implications for atherosclerotic plaque stab...

Por Rajagopalan, S, Meng, X P, Ramasamy, S, Harrison, D G, Galis, Z S
Publicado em 1996

Vascular expression of extracellular superoxide dismutase in atherosclerosis.

Por Fukai, T, Galis, Z S, Meng, X P, Parthasarathy, S, Harrison, D G
Publicado em 1998

Evidence for enhanced vascular superoxide anion production in nitrate tolerance. A novel mechanism underlying tolerance and cross-tolerance.

Por Münzel, T, Sayegh, H, Freeman, B A, Tarpey, M M, Harrison, D G
Publicado em 1995

Restoration of endothelium-dependent relaxation by dietary treatment of atherosclerosis.

Por Harrison, D G, Armstrong, M L, Freiman, P C, Heistad, D D
Publicado em 1987

Nitroglycerin metabolism in vascular tissue: role of glutathione S-transferases and relationship between NO. and NO2- formation.

Por Kurz, M A, Boyer, T D, Whalen, R, Peterson, T E, Harrison, D G
Publicado em 1993

Nitric oxide regulates vascular cell adhesion molecule 1 gene expression and redox-sensitive transcriptional events in human vascular endothelial cells.

Por Khan, B V, Harrison, D G, Olbrych, M T, Alexander, R W, Medford, R M
Publicado em 1996

Diet-induced atherosclerosis increases the release of nitrogen oxides from rabbit aorta.

Por Minor, R L, Myers, P R, Guerra, R, Bates, J N, Harrison, D G
Publicado em 1990

Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone.

Por Rajagopalan, S, Kurz, S, Münzel, T, Tarpey, M, Freeman, B A, Griendling, K K, Harrison, D G
Publicado em 1996

Differential cell-matrix mechanoadaptations and inflammation drive regional propensities to aortic fibrosis, aneurysm or dissection in hypertension

Por Bersi, M. R., Khosravi, R., Wujciak, A. J., Harrison, D. G., Humphrey, J. D.
Publicado no J R Soc Interface (2017)

Hydralazine prevents nitroglycerin tolerance by inhibiting activation of a membrane-bound NADH oxidase. A new action for an old drug.

Por Münzel, T, Kurz, S, Rajagopalan, S, Thoenes, M, Berrington, W R, Thompson, J A, Freeman, B A, Harrison, D G
Publicado em 1996

Interactions between L-arginine and L-glutamine change endothelial NO production. An effect independent of NO synthase substrate availability.

Por Arnal, J F, Münzel, T, Venema, R C, James, N L, Bai, C L, Mitch, W E, Harrison, D G
Publicado em 1995

Molecular cloning and characterization of the constitutive bovine aortic endothelial cell nitric oxide synthase.

Por Nishida, K, Harrison, D G, Navas, J P, Fisher, A A, Dockery, S P, Uematsu, M, Nerem, R M, Alexander, R W, Murphy, T J
Publicado em 1992