Regulation of expression of the lung amiloride-sensitive Na+ channel by steroid hormones.

Por Champigny, G, Voilley, N, Lingueglia, E, Friend, V, Barbry, P, Lazdunski, M
Publicado em 1994

CFTR fails to inhibit the epithelial sodium channel ENaC expressed in Xenopus laevis oocytes

Por Nagel, G, Barbry, P, Chabot, H, Brochiero, E, Hartung, K, Grygorczyk, R
Publicado em 2005

Novel Role for CFTR in Fluid Absorption from the Distal Airspaces of the Lung

Por Fang, X., Fukuda, N., Barbry, P., Sartori, C., Verkman, A.S., Matthay, M.A.
Publicado em 2002

The lung amiloride-sensitive Na+ channel: biophysical properties, pharmacology, ontogenesis, and molecular cloning.

Por Voilley, N, Lingueglia, E, Champigny, G, Mattéi, M G, Waldmann, R, Lazdunski, M, Barbry, P
Publicado em 1994

Purification and subunit structure of the [3H]phenamil receptor associated with the renal apical Na+ channel.

Por Barbry, P, Chassande, O, Vigne, P, Frelin, C, Ellory, C, Cragoe, E J, Lazdunski, M
Publicado em 1987

CDC25A targeting by miR-483-3p decreases CCND–CDK4/6 assembly and contributes to cell cycle arrest

Por Bertero, T, Gastaldi, C, Bourget-Ponzio, I, Mari, B, Meneguzzi, G, Barbry, P, Ponzio, G, Rezzonico, R
Publicado em 2013

Human kidney amiloride-binding protein: cDNA structure and functional expression.

Por Barbry, P, Champe, M, Chassande, O, Munemitsu, S, Champigny, G, Lingueglia, E, Maes, P, Frelin, C, Tartar, A, Ullrich, A
Publicado em 1990

A change in gating mode leading to increased intrinsic Cl- channel activity compensates for defective processing in a cystic fibrosis mutant corresponding to a mild form of the dis...

Por Champigny, G, Imler, J L, Puchelle, E, Dalemans, W, Gribkoff, V, Hinnrasky, J, Dott, K, Barbry, P, Pavirani, A, Lazdunski, M
Publicado em 1995

MiR-210 promotes a hypoxic phenotype and increases radioresistance in human lung cancer cell lines

Por Grosso, S, Doyen, J, Parks, S K, Bertero, T, Paye, A, Cardinaud, B, Gounon, P, Lacas-Gervais, S, Noël, A, Pouysségur, J, Barbry, P, Mazure, N M, Mari, B
Publicado em 2013

miR-210 is overexpressed in late stages of lung cancer and mediates mitochondrial alterations associated with modulation of HIF-1 activity

Por Puisségur, M-P, Mazure, N M, Bertero, T, Pradelli, L, Grosso, S, Robbe-Sermesant, K, Maurin, T, Lebrigand, K, Cardinaud, B, Hofman, V, Fourre, S, Magnone, V, Ricci, J E, Pouysségur, J, Gounon, P, Hofman, P, Barbry, P, Mari, B
Publicado em 2011