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Activation of cAMP-dependent protein kinase is required for heterologous desensitization of adenylyl cyclase in S49 wild-type lymphoma cells.
Molecular cloning of a rat testis form of the inhibitor protein of cAMP-dependent protein kinase.

Associative conditioning analog selectively increases cAMP levels of tail sensory neurons in Aplysia.
Phosphorylated Mr 32,000 dopamine- and cAMP-regulated phosphoprotein inhibits Na+,K(+)-ATPase activity in renal tubule cells.

Mechanism of regulation of the 422(aP2) gene by cAMP during preadipocyte differentiation.
Adrenocorticotropin (ACTH) induces phosphorylation of a cytoplasmic protein in intact isolated adrenocortical cells.

Stimulation of amyloid precursor protein synthesis by adrenergic receptors coupled to cAMP formation
Inactivation of S-adenosyl-L-homocysteine hydrolase by cAMP results from dissociation of enzyme-bound NAD+.

A beta-adrenergic receptor kinase-like enzyme is involved in olfactory signal termination.
A retinoic acid-responsive element is present in the 5' flanking region of the laminin B1 gene.

Structure of a lipid A phosphoethanolamine transferase suggests how conformational changes govern substrate binding
Sensitizing stimuli cause translocation of protein kinase C in Aplysia sensory neurons.

Phosphorylation/dephosphorylation of the beta-adrenergic receptor regulates its functional coupling to adenylate cyclase and subcellular distribution.
Ca2+ and cyclic AMP regulate phosphorylation of same two membrane-associated proteins specific to nerve tissue.

Human T-cell lymphotropic virus type I (HTLV-I) transcriptional activator, Tax, enhances CREB binding to HTLV-I 21-base-pair repeats by protein-protein interaction.
The phosphodiesterase inhibitor rolipram delivered after a spinal cord lesion promotes axonal regeneration and functional recovery

p300 is a component of an estrogen receptor coactivator complex.
In vitro desensitization of isolated nephron segments to vasopressin.

Activation of muscarinic acetylcholine receptors via their allosteric binding sites.
Cell-free transcription directed by the 422 adipose P2 gene promoter: activation by the CCAAT/enhancer binding protein.

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