Endotoxin-Induced Lung Inflammation Is Independent of the Complement Membrane Attack Complex

Several products of the activated complement system are known to modulate endothelial cell function in vitro. It has been shown that the membrane attack complex (MAC) (C5b-C9) can enhance tumor necrosis factor alpha (TNF-α)-induced expression of P- and E-selectin and intercellular adhesion molecule...

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Detalles Bibliográficos

Publicado en:	Infect Immun
Principais autores:	Brauer, R. B., Gegenfurtner, C., Neumann, B., Stadler, M., Heidecke, C. D., Holzmann, B.
Formato:	Artigo
Idioma:	Inglês
Publicado:	American Society for Microbiology (ASM) 2000
Assuntos:	Host Response and Inflammation
Acceso en liña:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC97323/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10678982/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.68.3.1626-1632.2000
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