Endotoxin-Induced Lung Inflammation Is Independent of the Complement Membrane Attack Complex

Several products of the activated complement system are known to modulate endothelial cell function in vitro. It has been shown that the membrane attack complex (MAC) (C5b-C9) can enhance tumor necrosis factor alpha (TNF-α)-induced expression of P- and E-selectin and intercellular adhesion molecule...

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Bibliografiske detaljer

Udgivet i:	Infect Immun
Principais autores:	Brauer, R. B., Gegenfurtner, C., Neumann, B., Stadler, M., Heidecke, C. D., Holzmann, B.
Format:	Artigo
Sprog:	Inglês
Udgivet:	American Society for Microbiology (ASM) 2000
Fag:	Host Response and Inflammation
Online adgang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC97323/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10678982/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.68.3.1626-1632.2000
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