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Loss of Annexin A7 Leads to Alterations in Frequency-Induced Shortening of Isolated Murine Cardiomyocytes

Annexin A7 has been proposed to function in the fusion of vesicles, acting as a Ca(2+) channel and as Ca(2+)-activated GTPase, thus inducing Ca(2+)/GTP-dependent secretory events. To understand the function of annexin A7, we have performed targeted disruption of the Anxa7 gene in mice. Matings betwe...

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Veröffentlicht in:Mol Cell Biol
Hauptverfasser: Herr, Claudia, Smyth, Neil, Ullrich, Susanne, Yun, Fan, Sasse, Phillip, Hescheler, Jürgen, Fleischmann, Bernd, Lasek, Katrin, Brixius, Klara, Schwinger, Robert H. G., Fässler, Reinhard, Schröder, Rolf, Noegel, Angelika A.
Format: Artigo
Sprache:Inglês
Veröffentlicht: Taylor & Francis 2001
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Online-Zugang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC87073/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11390641/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.21.13.4119-4128.2001
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