Loss of Annexin A7 Leads to Alterations in Frequency-Induced Shortening of Isolated Murine Cardiomyocytes

Annexin A7 has been proposed to function in the fusion of vesicles, acting as a Ca(2+) channel and as Ca(2+)-activated GTPase, thus inducing Ca(2+)/GTP-dependent secretory events. To understand the function of annexin A7, we have performed targeted disruption of the Anxa7 gene in mice. Matings betwe...

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Autores principales: Herr, Claudia, Smyth, Neil, Ullrich, Susanne, Yun, Fan, Sasse, Phillip, Hescheler, Jürgen, Fleischmann, Bernd, Lasek, Katrin, Brixius, Klara, Schwinger, Robert H. G., Fässler, Reinhard, Schröder, Rolf, Noegel, Angelika A.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Microbiology 2001
Materias:
Mammalian Genetic Models with Minimal or Complex Phenotypes
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC87073/
https://ncbi.nlm.nih.gov/pubmed/11390641
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.21.13.4119-4128.2001
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