Activation of p38 Mitogen-Activated Protein Kinase and c-Jun NH(2)-Terminal Kinase by Double-Stranded RNA and Encephalomyocarditis Virus: Involvement of RNase L, Protein Kinase R, and Alternative Pathways

Registos relacionados

• Mitogen-activated protein kinase kinase 7 is an activator of the c-Jun NH(2)-terminal kinase
  Por: Tournier, Cathy, et al.
  Publicado em: (1997)
• Anergic T cells are defective in both jun NH2-terminal kinase and mitogen-activated protein kinase signaling pathways
  Publicado em: (1996)
• Different Protein Tyrosine Kinases Are Required for B Cell Antigen Receptor–mediated Activation of Extracellular Signal–Regulated kinase, c-Jun NH(2)-terminal Kinase 1, and p38 Mitogen-activated Protein Kinase
  Por: Jiang, Aimin, et al.
  Publicado em: (1998)
• Activation of NF-κB by Double-Stranded RNA (dsRNA) in the Absence of Protein Kinase R and RNase L Demonstrates the Existence of Two Separate dsRNA-Triggered Antiviral Programs
  Por: Iordanov, Mihail S., et al.
  Publicado em: (2001)
• Encephalomyocarditis Virus Induces PKR-Independent Mitogen-Activated Protein Kinase Activation in Macrophages
  Por: Moran, Jason M., et al.
  Publicado em: (2005)