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Regulation of a Senescence Checkpoint Response by the E2F1 Transcription Factor and p14(ARF) Tumor Suppressor
Normal cells do not divide indefinitely due to a process known as replicative senescence. Human cells arrest growth with a senescent phenotype when they acquire one or more critically short telomeres as a consequence of cell division. Recent evidence suggests that certain types of DNA damage, chroma...
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| Main Authors: | , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
American Society for Microbiology
2000
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC85083/ https://ncbi.nlm.nih.gov/pubmed/10594030 |
| Clibeanna: |
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