Bcr-Abl with an SH3 Deletion Retains the Ability To Induce a Myeloproliferative Disease in Mice, yet c-Abl Activated by an SH3 Deletion Induces Only Lymphoid Malignancy

Lignende værker

• The NH(2)-Terminal Coiled-Coil Domain and Tyrosine 177 Play Important Roles in Induction of a Myeloproliferative Disease in Mice by Bcr-Abl
  af: Zhang, Xiaowu, et al.
  Udgivet: (2001)
• IL-3 receptor signaling is dispensable for BCR-ABL-induced myeloproliferative disease
  af: Wong, Stephane, et al.
  Udgivet: (2003)
• Expression of Interferon Consensus Sequence Binding Protein (ICSBP) Is Downregulated in Bcr-Abl-Induced Murine Chronic Myelogenous Leukemia-Like Disease, and Forced Coexpression of ICSBP Inhibits Bcr-Abl-Induced Myeloproliferative Disorder
  af: Hao, Sheryl X., et al.
  Udgivet: (2000)
• Mutational Analysis of SH2B3 in Korean Patients With BCR-ABL1 Negative Myeloproliferative Neoplasm
  af: Kim, Namhee, et al.
  Udgivet: (2016)
• Autoinhibition of Bcr-Abl through Its SH3 Domain
  af: Smith, Kristen M., et al.
  Udgivet: (2003)