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SPOP negatively regulates Toll-like receptor-induced inflammation by disrupting MyD88 self-association
Toll-like receptor (TLR) signaling pathways need to be tightly controlled to avoid excessive inflammation and unwanted damage to the host. Myeloid differentiation primary response gene 88 (MyD88) is a critical adaptor of TLR signaling. Here, we identified the speckle-type POZ protein (SPOP) as a MyD...
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| Veröffentlicht in: | Cell Mol Immunol |
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| Hauptverfasser: | , , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Nature Publishing Group UK
2020
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8245473/ https://ncbi.nlm.nih.gov/pubmed/32235916 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41423-020-0411-1 |
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