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Inactivation of Presenilin in inhibitory neurons results in decreased GABAergic responses and enhanced synaptic plasticity

Mutations in the Presenilin genes are the major genetic cause of Alzheimer's disease (AD). Presenilin (PS) is highly expressed in the hippocampus, which is particularly vulnerable in AD. Previous studies of PS function in the hippocampus, however, focused exclusively on excitatory neurons. Whet...

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Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:Mol Brain
Egile Nagusiak: Lee, Sang Hun, Bolshakov, Vadim Y., Shen, Jie
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: BioMed Central 2021
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC8152317/
https://ncbi.nlm.nih.gov/pubmed/34034776
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13041-021-00796-5
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