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Glia-selective deletion of complement C1q prevents radiation-induced cognitive deficits and neuroinflammation
The adverse neurocognitive sequelae following clinical radiation therapy (RT) for CNS malignancies are often long-lasting without any clinical recourse. Despite recent progress, the cellular mechanisms mediating RT-induced cognitive deficits (RICD) are poorly understood. The complement system is an...
Tallennettuna:
| Julkaisussa: | Cancer Res |
|---|---|
| Päätekijät: | , , , , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2020
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8137520/ https://ncbi.nlm.nih.gov/pubmed/33323383 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-20-2565 |
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