Glia-selective deletion of complement C1q prevents radiation-induced cognitive deficits and neuroinflammation

The adverse neurocognitive sequelae following clinical radiation therapy (RT) for CNS malignancies are often long-lasting without any clinical recourse. Despite recent progress, the cellular mechanisms mediating RT-induced cognitive deficits (RICD) are poorly understood. The complement system is an...

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Vydáno v:Cancer Res
Hlavní autoři: Markarian, Mineh, Krattli, Robert P., Baddour, Jabra D., Alikhani, Leila, Giedzinski, Erich, Usmani, Manal T., Agrawal, Anshu, Baulch, Janet E., Tenner, Andrea J., Acharya, Munjal M.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2020
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC8137520/
https://ncbi.nlm.nih.gov/pubmed/33323383
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-20-2565
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