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Gain of PITRM1 peptidase in cortical neurons affords protection of mitochondrial and synaptic function in an advanced age mouse model of Alzheimer’s disease

Mitochondrial dysfunction is one of the early pathological features of Alzheimer's disease (AD). Accumulation of cerebral and mitochondrial Aβ links to mitochondrial and synaptic toxicity. We have previously demonstrated the mechanism by which presequence peptidase (PITRM1)‐mediated clearance o...

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Vydáno v:Aging Cell
Hlavní autoři: Du, Fang, Yu, Qing, Yan, Shijun, Zhang, Zhihua, Vangavaragu, Jhansi Rani, Chen, Doris, Yan, Shi Fang, Yan, Shirley ShiDu
Médium: Artigo
Jazyk:Inglês
Vydáno: John Wiley and Sons Inc. 2021
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC8135081/
https://ncbi.nlm.nih.gov/pubmed/33951271
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.13368
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